Northwestern Medicine scientists have uncovered a new mechanism by which mutations in a gene parkin contribute to familial forms of Parkinson's disease. The discovery opens a new avenue for Parkinson's therapeutics, scientists report in a new study. The Northwestern scientists discovered that mutations in parkin result in a breakdown of contacts between two key workers in the cell -- lysosomes and mitochondria. Mitochondria are the main producers of energy in cells, and lysosomes recycle cellular debris that accumulates during normal function of our cells.

https://www.miragenews.com/therapeutic-target-for-parkinsons-disease-1050353/

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